graphic
  graphic Biomimetic Devices Lab  
  graphic 165 Packard Avenue, Dana 024, Medford, MA 02155 * Lab Phone: (617) 381-4403 * Office Phone: (617) 627-4196  
 

Publications:

Alice Witsell, D.P. Kane, S. Rubin, and M. McVey. 2009. Removal of the Bloom Syndrome DNA helicase extends the utility of imprecise transposon excision for making null mutations in Drosophila. Genetics, in press. [PDF ahead of print]

McVey, M. and S.E. Lee. 2008. MMEJ repair of double-strand breaks (director's cut): deleted sequences and alternative endings. Trends in Genetics 24: 529-538.[PDF] [Cover]

McVey, M., S. Andersen, G.J. Broze, and J.J. Sekelsky. 2007. Multiple roles for the Drosophila DmBlm helicase in maintenance of genome stability. Genetics 176: 1979-1992. [PDF] [Supplementary Data]

Rybarczyk, B.J., A.T. Baines, M. McVey, J.T. Thompson, and H.R. Wilkins. 2007. A case-based approach increases student learning outcomes and comprehension of cellular respiration concepts. Biochemistry and Molecular Biology Education 35: 181-186. [PDF]

McVey, M., D. Radut, and J.J. Sekelsky. 2004. End-joining repair of double-strand breaks in Drosophila is largely DNA ligase IV independent. Genetics 168: 2067-2076. [PDF]

McVey, M., J.R. LaRocque, M.D. Adams, and J.J. Sekelsky. 2004. Formation of deletions during double strand break repair in Drosophila DmBlm mutants occurs after strand invasion. Proc. Natl. Acad. Sci. USA 101: 15694-15699. [PDF]

McVey, M., M.D. Adams, J. LaRocque, and J.J. Sekelsky. 2004. Evidence for multiple cycles of strand invasion during repair of double-strand gaps in Drosophila. Genetics 167: 699-705. [PDF]

Baines, A.T., McVey, M., Rybarczyk, B., Thompson, J.T., and Wilkins, H.R. 2004. The mystery of the toxic flea dip: an interactive approach to teaching aerobic cellular respiration. Cell Biology Education 3: 62-68. [PDF]

Adams, M.D., M. McVey (co-first author), and J.J. Sekelsky. 2003. Drosophila BLM in double-strand break repair by synthesis-dependent strand annealing. Science 299: 265-267. [PDF]

McVey, M., M. Kaeberlein, H.A. Tissenbaum, and L. Guarente. 2001. The short life span of Saccharomyces cerevisiae sgs1 and srs2 mutants is a composite of normal aging processes and mitotic arrest due to defective recombination. Genetics 157: 1531-1542. [PDF]

Johnson, F.B., R.A. Marciniak, M. McVey, S.A. Stewart, W.C. Hahn, and L. Guarente. 2001. The Saccharomyces cerevisiae WRN homologue Sgs1p participates in telomere maintenance in cells lacking telomerase. EMBO Journal 20: 905-913. [PDF]

Stone, E.M., C. Reifsnyder, M. McVey, B. Gazo, and L. Pillus. 2000. Two classes of sir3 mutants enhance the sir1 mutant mating defect and abolish telomeric silencing in Saccharomyces cerevisiae. Genetics 155: 509-522. [PDF]

Kaeberlein, M., M. McVey (co-first author), and L. Guarente. 1999. SIR2/3/4 and SIR2 alone regulate life span in Saccharomyces cerevisiae by two different mechanisms. Genes and Development 13: 2570-80. [PDF]

 

  Tufts seal
  © Copyright 2006, Department of Biology, Tufts University. All rights reserved. Site designed by ITS.